G-Protein-Coupled Receptor Kinases
"G-Protein-Coupled Receptor Kinases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A family of serine-threonine kinases that are specific for G-PROTEIN-COUPLED RECEPTORS. They are regulatory proteins that play a role in G-protein-coupled receptor densensitization.
Descriptor ID |
D054768
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MeSH Number(s) |
D08.811.913.696.620.682.700.364 D12.644.360.293 D12.776.476.293
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Concept/Terms |
G-Protein-Coupled Receptor Kinases- G-Protein-Coupled Receptor Kinases
- G Protein Coupled Receptor Kinases
- Kinases, G-Protein-Coupled Receptor
- Receptor Kinases, G-Protein-Coupled
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Below are MeSH descriptors whose meaning is more general than "G-Protein-Coupled Receptor Kinases".
Below are MeSH descriptors whose meaning is more specific than "G-Protein-Coupled Receptor Kinases".
This graph shows the total number of publications written about "G-Protein-Coupled Receptor Kinases" by people in this website by year, and whether "G-Protein-Coupled Receptor Kinases" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2010 | 0 | 1 | 1 | 2012 | 1 | 0 | 1 |
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Below are the most recent publications written about "G-Protein-Coupled Receptor Kinases" by people in Profiles.
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Tilley DG, Zhu W, Myers VD, Barr LA, Gao E, Li X, Song J, Carter RL, Makarewich CA, Yu D, Troupes CD, Grisanti LA, Coleman RC, Koch WJ, Houser SR, Cheung JY, Feldman AM. ?-adrenergic receptor-mediated cardiac contractility is inhibited via vasopressin type 1A-receptor-dependent signaling. Circulation. 2014 Nov 11; 130(20):1800-11.
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Kamal FA, Travers JG, Blaxall BC. G protein-coupled receptor kinases in cardiovascular disease: why "where" matters. Trends Cardiovasc Med. 2012 Nov; 22(8):213-9.
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Khan SY, McLaughlin NJ, Kelher MR, Eckels P, Gamboni-Robertson F, Banerjee A, Silliman CC. Lysophosphatidylcholines activate G2A inducing G(ai)?1-/G(aq/)11- Ca?(+) flux, G(??)-Hck activation and clathrin/?-arrestin-1/GRK6 recruitment in PMNs. Biochem J. 2010 Nov 15; 432(1):35-45.
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