 Inflammation Mediators
"Inflammation Mediators" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The endogenous compounds that mediate inflammation (AUTACOIDS) and related exogenous compounds including the synthetic prostaglandins (PROSTAGLANDINS, SYNTHETIC).
| Descriptor ID |
D018836
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| MeSH Number(s) |
D23.469
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| Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Inflammation Mediators".
Below are MeSH descriptors whose meaning is more specific than "Inflammation Mediators".
This graph shows the total number of publications written about "Inflammation Mediators" by people in this website by year, and whether "Inflammation Mediators" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 0 | 1 | 1 | | 1997 | 2 | 1 | 3 | | 1998 | 3 | 1 | 4 | | 1999 | 2 | 3 | 5 | | 2000 | 1 | 0 | 1 | | 2001 | 0 | 1 | 1 | | 2002 | 0 | 1 | 1 | | 2003 | 1 | 5 | 6 | | 2004 | 5 | 3 | 8 | | 2005 | 4 | 4 | 8 | | 2006 | 8 | 8 | 16 | | 2007 | 2 | 4 | 6 | | 2008 | 9 | 6 | 15 | | 2009 | 8 | 8 | 16 | | 2010 | 4 | 12 | 16 | | 2011 | 9 | 7 | 16 | | 2012 | 8 | 20 | 28 | | 2013 | 8 | 15 | 23 | | 2014 | 3 | 9 | 12 | | 2015 | 8 | 9 | 17 | | 2016 | 6 | 13 | 19 | | 2017 | 6 | 14 | 20 | | 2018 | 8 | 8 | 16 | | 2019 | 6 | 14 | 20 | | 2020 | 5 | 5 | 10 | | 2021 | 2 | 10 | 12 | | 2022 | 1 | 1 | 2 | | 2023 | 1 | 0 | 1 | | 2024 | 0 | 1 | 1 |
To return to the timeline, click here.
Below are the most recent publications written about "Inflammation Mediators" by people in Profiles.
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Kant R, Mishra N, Kandhari K, Saba L, Michel C, Reisdorph R, Tewari-Singh N, Pantcheva MB, Petrash JM, Agarwal C, Agarwal R. Dexamethasone targets actin cytoskeleton signaling and inflammatory mediators to reverse sulfur mustard-induced toxicity in rabbit corneas. Toxicol Appl Pharmacol. 2024 02; 483:116834.
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Robinson PZ, Frank DN, Ramakrishnan VR. Inflammation resolution and specialized pro-resolving lipid mediators in chronic rhinosinusitis. Expert Rev Clin Immunol. 2023 Jul-Dec; 19(8):969-979.
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Tufa A, Gebremariam TH, Manyazewal T, Getinet T, Webb DL, Hellstr?m PM, Genet S. Inflammatory mediators profile in patients hospitalized with COVID-19: A comparative study. Front Immunol. 2022; 13:964179.
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Cervantes-Alvarez E, la Rosa NL, la Mora MS, Valdez-Sandoval P, Palacios-Jimenez M, Rodriguez-Alvarez F, Vera-Maldonado BI, Aguirre-Aguilar E, Escobar-Valderrama JM, Alanis-Mendizabal J, M?ndez-Guerrero O, Tejeda-Dominguez F, Torres-Ru?z J, G?mez-Mart?n D, Colborn KL, Kershenobich D, Huang CA, Navarro-Alvarez N. Galectin-3 as a potential prognostic biomarker of severe COVID-19 in SARS-CoV-2 infected patients. Sci Rep. 2022 02 03; 12(1):1856.
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Zhang P, The E, Luo Z, Zhai Y, Yao Q, Ao L, Fullerton DA, Xu D, Meng X. Pro-inflammatory mediators released by activated monocytes promote aortic valve fibrocalcific activity. Mol Med. 2022 01 21; 28(1):5.
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Sabaawy HE, Ryan BM, Khiabanian H, Pine SR. JAK/STAT of all trades: linking inflammation with cancer development, tumor progression and therapy resistance. Carcinogenesis. 2021 12 31; 42(12):1411-1419.
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Montford JR, Bauer C, Rahkola J, Reisz JA, Floyd D, Hopp K, Soranno DE, Klawitter J, Weiser-Evans MCM, Nemenoff R, Faubel S, Furgeson SB. 15-Lipoxygenase worsens renal fibrosis, inflammation, and metabolism in a murine model of ureteral obstruction. Am J Physiol Renal Physiol. 2022 01 01; 322(1):F105-F119.
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Bantle CM, Rocha SM, French CT, Phillips AT, Tran K, Olson KE, Bass TA, Aboellail T, Smeyne RJ, Tjalkens RB. Astrocyte inflammatory signaling mediates a-synuclein aggregation and dopaminergic neuronal loss following viral encephalitis. Exp Neurol. 2021 12; 346:113845.
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Hobby ARH, Berretta RM, Eaton DM, Kubo H, Feldsott E, Yang Y, Headrick AL, Koch KA, Rubino M, Kurian J, Khan M, Tan Y, Mohsin S, Gallucci S, McKinsey TA, Houser SR. Cortical bone stem cells modify cardiac inflammation after myocardial infarction by inducing a novel macrophage phenotype. Am J Physiol Heart Circ Physiol. 2021 10 01; 321(4):H684-H701.
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Chang JC, Matsubara D, Morgan RW, Diorio C, Nadaraj S, Teachey DT, Bassiri H, Behrens EM, Banerjee A. Skewed Cytokine Responses Rather Than the Magnitude of the Cytokine Storm May Drive Cardiac Dysfunction in Multisystem Inflammatory Syndrome in Children. J Am Heart Assoc. 2021 08 17; 10(16):e021428.
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