Mice, Inbred AKR
"Mice, Inbred AKR" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An inbred strain of mouse that is widely used in IMMUNOLOGY studies and cancer research.
Descriptor ID |
D008806
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MeSH Number(s) |
B01.050.050.199.520.520.318 B01.050.150.900.649.313.992.635.505.500.400.318
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Concept/Terms |
Mice, Inbred AKR- Mice, Inbred AKR
- AKR Mice, Inbred
- Inbred AKR Mice
- Mouse, Inbred AKR
- AKR Mouse, Inbred
- Inbred AKR Mouse
- Mice, AKR
- AKR Mice
- Mouse, AKR
- AKR Mouse
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Below are MeSH descriptors whose meaning is more general than "Mice, Inbred AKR".
Below are MeSH descriptors whose meaning is more specific than "Mice, Inbred AKR".
This graph shows the total number of publications written about "Mice, Inbred AKR" by people in this website by year, and whether "Mice, Inbred AKR" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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1996 | 0 | 1 | 1 | 1997 | 0 | 1 | 1 | 2003 | 0 | 2 | 2 | 2008 | 0 | 1 | 1 | 2009 | 0 | 2 | 2 | 2011 | 0 | 1 | 1 |
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Below are the most recent publications written about "Mice, Inbred AKR" by people in Profiles.
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Vang A, Clements RT, Chichger H, Kue N, Allawzi A, O'Connell K, Jeong EM, Dudley SC, Sakhatskyy P, Lu Q, Zhang P, Rounds S, Choudhary G. Effect of a7 nicotinic acetylcholine receptor activation on cardiac fibroblasts: a mechanism underlying RV fibrosis associated with cigarette smoke exposure. Am J Physiol Lung Cell Mol Physiol. 2017 05 01; 312(5):L748-L759.
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Podowski M, Calvi C, Metzger S, Misono K, Poonyagariyagorn H, Lopez-Mercado A, Ku T, Lauer T, McGrath-Morrow S, Berger A, Cheadle C, Tuder R, Dietz HC, Mitzner W, Wise R, Neptune E. Angiotensin receptor blockade attenuates cigarette smoke-induced lung injury and rescues lung architecture in mice. J Clin Invest. 2012 Jan; 122(1):229-40.
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Ghosh S, Thakur MK. Interaction of estrogen receptor-alpha ligand binding domain with nuclear proteins of aging mouse brain. J Neurosci Res. 2009 Aug 15; 87(11):2591-600.
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Ge XN, Chu HW, Minor MN, Case SR, Bosch DG, Martin RJ. Roflumilast increases Clara cell secretory protein in cigarette smoke-exposed mice. COPD. 2009 Jun; 6(3):185-91.
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Ghosh S, Thakur MK. Interaction of estrogen receptor-alpha transactivation domain with nuclear proteins of mouse brain: p68 RNA helicase shows age- and sex-specific change. J Neurosci Res. 2009 May 01; 87(6):1323-8.
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Zaas AK, Liao G, Chien JW, Weinberg C, Shore D, Giles SS, Marr KA, Usuka J, Burch LH, Perera L, Perfect JR, Peltz G, Schwartz DA. Plasminogen alleles influence susceptibility to invasive aspergillosis. PLoS Genet. 2008 Jun 20; 4(6):e1000101.
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Butt CM, King NM, Stitzel JA, Collins AC. Interaction of the nicotinic cholinergic system with ethanol withdrawal. J Pharmacol Exp Ther. 2004 Feb; 308(2):591-9.
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Butt CM, Hutton SR, Stitzel JA, Balogh SA, Owens JC, Collins AC. A polymorphism in the alpha4 nicotinic receptor gene (Chrna4) modulates enhancement of nicotinic receptor function by ethanol. Alcohol Clin Exp Res. 2003 May; 27(5):733-42.
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McKeever U, Khandekar S, Jesson M, Newcomb J, Gregory P, Naylor J, Haskins K, Jones B. Maternal immunization with a soluble TCR-Ig chimeric protein: long term, V beta-8 family-specific suppression of T cells by maternally transferred antibodies. J Immunol. 1997 Dec 15; 159(12):5936-45.
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Brady KP, Rowe LB, Her H, Stevens TJ, Eppig J, Sussman DJ, Sikela J, Beier DR. Genetic mapping of 262 loci derived from expressed sequences in a murine interspecific cross using single-strand conformational polymorphism analysis. Genome Res. 1997 Nov; 7(11):1085-93.
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