 Joe Nassour
| Title | Asst Professor |
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| Institution | University of Colorado Denver - Anschutz Medical Campus |
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| Department | SOM-BioChem&Molecular Genetics |
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 Bibliographic
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Nassour J, Przetocka S, Karlseder J. Telomeres as hotspots for innate immunity and inflammation. DNA Repair (Amst). 2024 Jan; 133:103591. PMID: 37951043.
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Moreno SP, Fust? JM, Kaiser M, Li JSZ, Nassour J, Haggblom C, Denchi EL, Karlseder J. TZAP overexpression induces telomere dysfunction and ALT-like activity in ATRX/DAXX-deficient cells. iScience. 2023 Apr 21; 26(4):106405. PMID: 37013192.
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Nassour J, Aguiar LG, Correia A, Schmidt TT, Mainz L, Przetocka S, Haggblom C, Tadepalle N, Williams A, Shokhirev MN, Akincilar SC, Tergaonkar V, Shadel GS, Karlseder J. Telomere-to-mitochondria signalling by ZBP1 mediates replicative crisis. Nature. 2023 02; 614(7949):767-773. PMID: 36755096.
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Mendez-Bermudez A, Lototska L, Pousse M, Tessier F, Croce O, Latrick CM, Cherdyntseva V, Nassour J, Xiaohua J, Lu Y, Abbadie C, Gagos S, Ye J, Gilson E. Selective pericentromeric heterochromatin dismantling caused by TP53 activation during senescence. Nucleic Acids Res. 2022 07 22; 50(13):7493-7510. PMID: 35819196.
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Goy E, Tomezak M, Facchin C, Martin N, Bouchaert E, Benoit J, de Schutter C, Nassour J, Saas L, Drullion C, Brodin PM, Vandeputte A, Molendi-Coste O, Pineau L, Goormachtigh G, Pluquet O, Pourtier A, Cleri F, Lartigau E, Penel N, Abbadie C. The out-of-field dose in radiation therapy induces delayed tumorigenesis by senescence evasion. Elife. 2022 03 18; 11. PMID: 35302491.
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Nassour J, Schmidt TT, Karlseder J. Telomeres and Cancer: Resolving the Paradox. Annu Rev Cancer Biol. 2021 Mar; 5(1):59-77. PMID: 34532611.
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Nassour J, Radford R, Correia A, Fust? JM, Schoell B, Jauch A, Shaw RJ, Karlseder J. Autophagic cell death restricts chromosomal instability during replicative crisis. Nature. 2019 01; 565(7741):659-663. PMID: 30675059.
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Paget S, Dubuissez M, Dehennaut V, Nassour J, Harmon BT, Spruyt N, Loison I, Abbadie C, Rood BR, Leprince D. HIC1 (hypermethylated in cancer 1) SUMOylation is dispensable for DNA repair but is essential for the apoptotic DNA damage response (DDR) to irreparable DNA double-strand breaks (DSBs). Oncotarget. 2017 Jan 10; 8(2):2916-2935. PMID: 27935866.
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Nassour J, Abbadie C. A novel role for DNA single-strand breaks in senescence and neoplastic escape of epithelial cells. Mol Cell Oncol. 2016; 3(5):e1190885. PMID: 27857969.
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Nassour J, Martien S, Martin N, Deruy E, Tomellini E, Malaquin N, Bouali F, Sabatier L, Wernert N, Pinte S, Gilson E, Pourtier A, Pluquet O, Abbadie C. Defective DNA single-strand break repair is responsible for senescence and neoplastic escape of epithelial cells. Nat Commun. 2016 Jan 29; 7:10399. PMID: 26822533.
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Deruy E, Nassour J, Martin N, Vercamer C, Malaquin N, Bertout J, Chelli F, Pourtier A, Pluquet O, Abbadie C. Level of macroautophagy drives senescent keratinocytes into cell death or neoplastic evasion. Cell Death Dis. 2014 Dec 18; 5:e1577. PMID: 25522271.
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Dehennaut V, Loison I, Dubuissez M, Nassour J, Abbadie C, Leprince D. DNA double-strand breaks lead to activation of hypermethylated in cancer 1 (HIC1) by SUMOylation to regulate DNA repair. J Biol Chem. 2013 Apr 12; 288(15):10254-64. PMID: 23417673.
This graph shows the total number of publications by year, by first, middle/unknown, or last author.
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| Year | Publications |
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| 2013 | 1 | | 2014 | 1 | | 2016 | 2 | | 2017 | 1 | | 2019 | 1 | | 2021 | 1 | | 2022 | 2 | | 2023 | 3 |
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