 ATP Citrate (pro-S)-Lyase
"ATP Citrate (pro-S)-Lyase" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An enzyme that, in the presence of ATP and COENZYME A, catalyzes the cleavage of citrate to yield acetyl CoA, oxaloacetate, ADP, and ORTHOPHOSPHATE. This reaction represents an important step in fatty acid biosynthesis. This enzyme was formerly listed as EC 4.1.3.8.
| Descriptor ID |
D001275
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| MeSH Number(s) |
D08.811.913.050.331
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| Concept/Terms |
ATP Citrate (pro-S)-Lyase- ATP Citrate (pro-S)-Lyase
- Citrate Cleavage Enzyme
- Cleavage Enzyme, Citrate
- ATP Citrate Synthase
- Citrate Synthase, ATP
- Synthase, ATP Citrate
- ATP-Dependent Citrate Lyase
- ATP Dependent Citrate Lyase
- Citrate Lyase, ATP-Dependent
- Lyase, ATP-Dependent Citrate
- ATP Citrate (pro-3S)-Lyase
- ATP Citrate Lyase
- Citrate Lyase, ATP
- Lyase, ATP Citrate
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Below are MeSH descriptors whose meaning is more general than "ATP Citrate (pro-S)-Lyase".
Below are MeSH descriptors whose meaning is more specific than "ATP Citrate (pro-S)-Lyase".
This graph shows the total number of publications written about "ATP Citrate (pro-S)-Lyase" by people in this website by year, and whether "ATP Citrate (pro-S)-Lyase" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2005 | 0 | 1 | 1 | | 2013 | 0 | 1 | 1 |
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Below are the most recent publications written about "ATP Citrate (pro-S)-Lyase" by people in Profiles.
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Prola A, Nichtova Z, Pires Da Silva J, Piquereau J, Monceaux K, Guilbert A, Gressette M, Ventura-Clapier R, Garnier A, Zahradnik I, Novotova M, Lemaire C. Endoplasmic reticulum stress induces cardiac dysfunction through architectural modifications and alteration of mitochondrial function in cardiomyocytes. Cardiovasc Res. 2019 02 01; 115(2):328-342.
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Chait A, Eckel RH. Lipids, Lipoproteins, and Cardiovascular Disease: Clinical Pharmacology Now and in the Future. J Clin Endocrinol Metab. 2016 Mar; 101(3):804-14.
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Schultz RL, Kullman EL, Waters RP, Huang H, Kirwan JP, Gerdes AM, Swallow JG. Metabolic adaptations of skeletal muscle to voluntary wheel running exercise in hypertensive heart failure rats. Physiol Res. 2013; 62(4):361-9.
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Jiang T, Liebman SE, Lucia MS, Li J, Levi M. Role of altered renal lipid metabolism and the sterol regulatory element binding proteins in the pathogenesis of age-related renal disease. Kidney Int. 2005 Dec; 68(6):2608-20.
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Pugazhenthi S, Hussain A, Yu B, Brownsey RW, Angel JF, Khandelwal RL. Vanadate induces normolipidemia and a reduction in the levels of hepatic lipogenic enzymes in obese Zucker rat. Mol Cell Biochem. 1995 Dec 6-20; 153(1-2):211-5.
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Avruch J, Alexander MC, Palmer JL, Pierce MW, Nemenoff RA, Blackshear PJ, Tipper JP, Witters LA. Role of insulin-stimulated protein phosphorylation in insulin action. Fed Proc. 1982 Aug; 41(10):2629-33.
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