 Glucose Transporter Type 1
"Glucose Transporter Type 1" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A ubiquitously expressed glucose transporter that is important for constitutive, basal GLUCOSE transport. It is predominately expressed in ENDOTHELIAL CELLS and ERYTHROCYTES at the BLOOD-BRAIN BARRIER and is responsible for GLUCOSE entry into the BRAIN.
| Descriptor ID |
D051272
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| MeSH Number(s) |
D12.776.157.530.500.500.500 D12.776.157.530.937.563.500 D12.776.543.585.500.500.500 D12.776.543.585.937.625.500
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| Concept/Terms |
Glucose Transporter Type 1- Glucose Transporter Type 1
- GLUT-1 Protein
- GLUT 1 Protein
- Solute Carrier Family 2, Facilitated Glucose Transporter, Member 1 Protein
- SLC2A1 Protein
- GLUT1 Protein
- Erythrocyte Glucose Transporter
- Glucose Transporter, Erythrocyte
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Below are MeSH descriptors whose meaning is more general than "Glucose Transporter Type 1".
Below are MeSH descriptors whose meaning is more specific than "Glucose Transporter Type 1".
This graph shows the total number of publications written about "Glucose Transporter Type 1" by people in this website by year, and whether "Glucose Transporter Type 1" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1996 | 0 | 1 | 1 | | 1999 | 0 | 1 | 1 | | 2001 | 0 | 1 | 1 | | 2004 | 0 | 1 | 1 | | 2006 | 1 | 0 | 1 | | 2007 | 0 | 2 | 2 | | 2009 | 1 | 1 | 2 | | 2010 | 0 | 1 | 1 | | 2011 | 1 | 0 | 1 | | 2012 | 2 | 0 | 2 | | 2014 | 1 | 0 | 1 | | 2016 | 1 | 1 | 2 | | 2017 | 1 | 0 | 1 | | 2018 | 1 | 0 | 1 | | 2019 | 1 | 2 | 3 | | 2020 | 2 | 0 | 2 | | 2021 | 1 | 0 | 1 | | 2022 | 0 | 1 | 1 | | 2023 | 0 | 1 | 1 |
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Below are the most recent publications written about "Glucose Transporter Type 1" by people in Profiles.
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Joshi N, Sahay A, Mane A, Sundrani D, Randhir K, Wagh G, Thornburg K, Powell T, Yajnik C, Joshi S. Altered expression of nutrient transporters in syncytiotrophoblast membranes in preeclampsia placentae. Placenta. 2023 08; 139:181-189.
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Lynch CS, Kennedy VC, Tanner AR, Ali A, Winger QA, Rozance PJ, Anthony RV. Impact of Placental SLC2A3 Deficiency during the First-Half of Gestation. Int J Mol Sci. 2022 Oct 19; 23(20).
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Li K, Ji X, Seeley R, Lee WC, Shi Y, Song F, Liao X, Song C, Huang X, Rux D, Cao J, Luo X, Anderson SM, Huang W, Long F. Impaired glucose metabolism underlies articular cartilage degeneration in osteoarthritis. FASEB J. 2022 06; 36(6):e22377.
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Dakic T, Lakic I, Zec M, Takic M, Stojiljkovic M, Jevdjovic T. Fructose-rich diet and walnut supplementation differently regulate rat hypothalamic and hippocampal glucose transporters expression. J Sci Food Agric. 2021 Nov; 101(14):5984-5991.
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Kramer AC, Steinhauser CB, Gao H, Seo H, McLendon BA, Burghardt RC, Wu G, Bazer FW, Johnson GA. Steroids Regulate SLC2A1 and SLC2A3 to Deliver Glucose Into Trophectoderm for Metabolism via Glycolysis. Endocrinology. 2020 08 01; 161(8).
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James-Allan LB, Teal S, Powell TL, Jansson T. Changes in Placental Nutrient Transporter Protein Expression and Activity Across Gestation in Normal and Obese Women. Reprod Sci. 2020 09; 27(9):1758-1769.
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Al-Saleem J, Dirksen WP, Martinez MP, Shkriabai N, Kvaratskhelia M, Ratner L, Green PL. HTLV-1 Tax-1 interacts with SNX27 to regulate cellular localization of the HTLV-1 receptor molecule, GLUT1. PLoS One. 2019; 14(3):e0214059.
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Michelsen TM, Holme AM, Holm MB, Roland MC, Haugen G, Powell TL, Jansson T, Henriksen T. Uteroplacental Glucose Uptake and Fetal Glucose Consumption: A Quantitative Study in Human Pregnancies. J Clin Endocrinol Metab. 2019 03 01; 104(3):873-882.
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Angione K, Eschbach K, Smith G, Joshi C, Demarest S. Genetic testing in a cohort of patients with potential epilepsy with myoclonic-atonic seizures. Epilepsy Res. 2019 02; 150:70-77.
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Kapur RP, Arnold MA, Conces MR, Ambartsumyan L, Avansino J, Levitt M, Wood R, Mast KJ. Remodeling of Rectal Innervation After Pullthrough Surgery for Hirschsprung Disease: Relevance to Criteria for the Determination of Retained Transition Zone. Pediatr Dev Pathol. 2019 Jul-Aug; 22(4):292-303.
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