 Mice, Inbred ICR
"Mice, Inbred ICR" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An inbred strain of mouse that is used as a general purpose research strain, for therapeutic drug testing, and for the genetic analysis of CARCINOGEN-induced COLON CANCER.
| Descriptor ID |
D008813
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| MeSH Number(s) |
B01.050.050.199.520.520.510 B01.050.150.900.649.313.992.635.505.500.400.510
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| Concept/Terms |
Mice, Inbred ICR- Mice, Inbred ICR
- ICR Mice, Inbred
- Inbred ICR Mice
- Mouse, Inbred ICR
- ICR Mouse, Inbred
- Inbred ICR Mouse
- Mice, ICR
- ICR Mice
- Mouse, ICR
- ICR Mouse
Mouse, Inbred ICRC- Mouse, Inbred ICRC
- ICRC Mouse, Inbred
- Inbred ICRC Mouse
- Mice, Inbred ICRC
- ICRC Mice, Inbred
- Inbred ICRC Mice
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Below are MeSH descriptors whose meaning is more general than "Mice, Inbred ICR".
Below are MeSH descriptors whose meaning is more specific than "Mice, Inbred ICR".
This graph shows the total number of publications written about "Mice, Inbred ICR" by people in this website by year, and whether "Mice, Inbred ICR" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1992 | 0 | 1 | 1 | | 1993 | 0 | 2 | 2 | | 1994 | 0 | 1 | 1 | | 1995 | 0 | 5 | 5 | | 1996 | 0 | 3 | 3 | | 1997 | 0 | 5 | 5 | | 1998 | 0 | 3 | 3 | | 1999 | 0 | 3 | 3 | | 2000 | 0 | 3 | 3 | | 2001 | 1 | 2 | 3 | | 2002 | 0 | 1 | 1 | | 2003 | 0 | 2 | 2 | | 2004 | 0 | 4 | 4 | | 2005 | 0 | 3 | 3 | | 2006 | 0 | 2 | 2 | | 2007 | 0 | 4 | 4 | | 2008 | 0 | 2 | 2 | | 2009 | 0 | 5 | 5 | | 2010 | 0 | 5 | 5 | | 2011 | 0 | 2 | 2 | | 2012 | 0 | 3 | 3 | | 2013 | 0 | 2 | 2 | | 2014 | 0 | 7 | 7 | | 2015 | 0 | 3 | 3 | | 2016 | 0 | 2 | 2 | | 2017 | 0 | 4 | 4 | | 2018 | 0 | 2 | 2 | | 2019 | 0 | 4 | 4 | | 2020 | 0 | 1 | 1 |
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Below are the most recent publications written about "Mice, Inbred ICR" by people in Profiles.
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Wright CJ, Cari EL, Sandoval J, Bales E, Sam PK, Zarate MA, Polotsky AJ, Kallen AN, Johnson J. Control of Murine Primordial Follicle Growth Activation by I?B/NF?B Signaling. Reprod Sci. 2020 11; 27(11):2063-2074.
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Sandoval J, Orlicky DJ, Allawzi A, Butler B, Ju C, Phan CT, Toston R, De Dios R, Nguyen L, McKenna S, Nozik-Grayck E, Wright CJ. Toxic Acetaminophen Exposure Induces Distal Lung ER Stress, Proinflammatory Signaling, and Emphysematous Changes in the Adult Murine Lung. Oxid Med Cell Longev. 2019; 2019:7595126.
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Lee W, Hiura LC, Yang E, Broekman KA, Ophir AG, Curley JP. Social status in mouse social hierarchies is associated with variation in oxytocin and vasopressin 1a receptor densities. Horm Behav. 2019 08; 114:104551.
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Nguyen L, Castro O, De Dios R, Sandoval J, McKenna S, Wright CJ. Sex-differences in LPS-induced neonatal lung injury. Sci Rep. 2019 06 11; 9(1):8514.
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Nguyen L, Sandoval J, De Dios R, Yihdego E, Zarate M, Castro O, McKenna S, Wright CJ. The hepatic innate immune response is lobe-specific in a murine model endotoxemia. Innate Immun. 2019 02; 25(2):144-154.
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McKenna S, Burey T, Sandoval J, Nguyen L, Castro O, Gudipati S, Gonzalez J, El Kasmi KC, Wright CJ. Immunotolerant p50/NF?B Signaling and Attenuated Hepatic IFNß Expression Increases Neonatal Sensitivity to Endotoxemia. Front Immunol. 2018; 9:2210.
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Xu F, Takahashi H, Tanaka Y, Ichinose S, Niwa S, Wicklund MP, Hirokawa N. KIF1Bß mutations detected in hereditary neuropathy impair IGF1R transport and axon growth. J Cell Biol. 2018 10 01; 217(10):3480-3496.
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Wang JS, Infante CR, Park S, Menke DB. PITX1 promotes chondrogenesis and myogenesis in mouse hindlimbs through conserved regulatory targets. Dev Biol. 2018 02 01; 434(1):186-195.
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McKenna S, Butler B, Jatana L, Ghosh S, Wright CJ. Inhibition of I?Bß/NF?B signaling prevents LPS-induced IL1ß expression without increasing apoptosis in the developing mouse lung. Pediatr Res. 2017 Dec; 82(6):1064-1072.
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Rosario FJ, Nathanielsz PW, Powell TL, Jansson T. Maternal folate deficiency causes inhibition of mTOR signaling, down-regulation of placental amino acid transporters and fetal growth restriction in mice. Sci Rep. 2017 06 21; 7(1):3982.
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