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Search Results to Heide L Ford

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Ford, Heide

One or more keywords matched the following items that are connected to Ford, Heide

Item Type	Name
Academic Article	Transcriptional control of the cell cycle in mammary gland development and tumorigenesis.
Academic Article	The Six1 homeoprotein stimulates tumorigenesis by reactivation of cyclin A1.
Academic Article	Gene amplification is a mechanism of Six1 overexpression in breast cancer.
Academic Article	Cell cycle regulation of the human Six1 homeoprotein is mediated by APC(Cdh1).
Academic Article	The six family of homeobox genes in development and cancer.
Academic Article	The Six1 homeoprotein induces human mammary carcinoma cells to undergo epithelial-mesenchymal transition and metastasis in mice through increasing TGF-beta signaling.
Academic Article	Epithelial-mesenchymal transition in development and cancer.
Academic Article	Homeoprotein Six1 increases TGF-beta type I receptor and converts TGF-beta signaling from suppressive to supportive for tumor growth.
Academic Article	TRAIL receptor signaling regulation of chemosensitivity in vivo but not in vitro.
Academic Article	Distinct TRAIL resistance mechanisms can be overcome by proteasome inhibition but not generally by synergizing agents.
Academic Article	On the TRAIL to successful cancer therapy? Predicting and counteracting resistance against TRAIL-based therapeutics.
Academic Article	EWS/FLI1 regulates EYA3 in Ewing sarcoma via modulation of miRNA-708, resulting in increased cell survival and chemoresistance.
Academic Article	Structure-function analyses of the human SIX1-EYA2 complex reveal insights into metastasis and BOR syndrome.
Academic Article	A mechanism of cyclin D1 action encoded in the patterns of gene expression in human cancer.
Academic Article	Six1 overexpression in ovarian carcinoma causes resistance to TRAIL-mediated apoptosis and is associated with poor survival.
Academic Article	Characterization of the Six1 homeobox gene in normal mammary gland morphogenesis.
Academic Article	Six1 overexpression in mammary cells induces genomic instability and is sufficient for malignant transformation.
Academic Article	Epithelial-mesenchymal transition in cancer: parallels between normal development and tumor progression.
Academic Article	Mammary gland studies as important contributors to the cause of epithelial mesenchymal plasticity in malignancy.
Academic Article	Biochemical and functional characterization of six SIX1 Branchio-oto-renal syndrome mutations.
Academic Article	Six1 expands the mouse mammary epithelial stem/progenitor cell pool and induces mammary tumors that undergo epithelial-mesenchymal transition.
Academic Article	SIX1 induces lymphangiogenesis and metastasis via upregulation of VEGF-C in mouse models of breast cancer.
Academic Article	Molecular pathways: targeting the TGF-? pathway for cancer therapy.
Academic Article	Breast cancer epithelial-to-mesenchymal transition: examining the functional consequences of plasticity.
Academic Article	Specificity and prognostic validation of a polyclonal antibody to detect Six1 homeoprotein in ovarian cancer.
Concept	Mammary Neoplasms, Experimental
Concept	Tumor Burden
Concept	Brain Neoplasms
Concept	Lung Neoplasms
Concept	Tumor Suppressor Protein p53
Concept	Neoplasm Metastasis
Concept	Genes, Tumor Suppressor
Concept	Biomarkers, Tumor
Concept	Ovarian Neoplasms
Concept	Pancreatic Neoplasms
Concept	Neoplasm Proteins
Concept	Drug Resistance, Neoplasm
Concept	Neoplasm Transplantation
Concept	Breast Neoplasms
Concept	Bone Neoplasms
Concept	Antibodies, Neoplasm
Concept	Tumor Cells, Cultured
Concept	Neoplasms
Concept	Cell Line, Tumor
Concept	Neoplasm Recurrence, Local
Concept	Cerebellar Neoplasms
Concept	Neoplasm Invasiveness
Concept	Eye Neoplasms
Academic Article	Epithelial-mesenchymal transition and tumor suppression are controlled by a reciprocal feedback loop between ZEB1 and Grainyhead-like-2.
Academic Article	CDK6 binds and promotes the degradation of the EYA2 protein.
Academic Article	Expression of Six1 in luminal breast cancers predicts poor prognosis and promotes increases in tumor initiating cells by activation of extracellular signal-regulated kinase and transforming growth factor-beta signaling pathways.
Academic Article	Homeoprotein Six2 promotes breast cancer metastasis via transcriptional and epigenetic control of E-cadherin expression.
Academic Article	Vascular endothelial growth factor C promotes breast cancer progression via a novel antioxidant mechanism that involves regulation of superoxide dismutase 3.
Academic Article	The SIX1-EYA transcriptional complex as a therapeutic target in cancer.
Academic Article	TWIST1-Induced miR-424 Reversibly Drives Mesenchymal Programming while Inhibiting Tumor Initiation.
Academic Article	The Six1 oncoprotein downregulates p53 via concomitant regulation of RPL26 and microRNA-27a-3p.
Academic Article	CD147: a small molecule transporter ancillary protein at the crossroad of multiple hallmarks of cancer and metabolic reprogramming.
Academic Article	Non-cell-autonomous Effects of Autophagy Inhibition in Tumor Cells Promote Growth of Drug-resistant Cells.
Academic Article	A Genome-Wide Loss-of-Function Screen Identifies SLC26A2 as a Novel Mediator of TRAIL Resistance.
Academic Article	Clonal cooperativity in heterogenous cancers.
Grant	Identify inhibitors of the Six1/Eya interaction using high throughput screening
Grant	Developing cancer therapies through targeting the Six1/Eya transcriptional complex
Grant	Role of Six1 and the miR106b 25 cluster in EMT and Tumor Progression
Grant	The anti-tumorigenic and anti-metastatic potential of Eya phosphatase inhibitors
Grant	TRAIL Receptor Signaling in Human Tumors
Grant	The Six1/Eya Transcriptional complex as a mediator of lymphangiogenesis and lymph
Grant	HSIX1 IN CANCER AND THE CELL CYCLE
Grant	Using zebrafish to study the role of six1a/b and microRNA-30a in rhabdomyosarcoma (RMS) initiation and progression
Grant	Identify inhibitors of the Eya phosphatase activity using high throughput screeni
Academic Article	EMT cells increase breast cancer metastasis via paracrine GLI activation in neighbouring tumour cells.
Academic Article	The Eya phosphatase: Its unique role in cancer.
Grant	2017 Mammary Gland Biology Gordon Research Conference & Gordon Research Seminar
Grant	Role of Eya3 in regulating the immune microenvironment to promote breast tumor progression
Academic Article	Eya3 partners with PP2A to induce c-Myc stabilization and tumor progression.
Academic Article	Eya3 promotes breast tumor-associated immune suppression via threonine phosphatase-mediated PD-L1 upregulation.
Grant	Examining the EYA2/MYC axis in Group 3 Medulloblastoma
Academic Article	Author Correction: Eya3 partners with PP2A to induce c-Myc stabilization and tumor progression.
Academic Article	Publisher Correction: EMT cells increase breast cancer metastasis via paracrine GLI activation in neighbouring tumour cells.
Academic Article	SIX2 Mediates Late-Stage Metastasis via Direct Regulation of SOX2 and Induction of a Cancer Stem Cell Program.
Grant	Deciphering Mechanisms by which Tumor Cells Collaborate to Mediate Metastasis
Concept	Tumor Microenvironment
Concept	Triple Negative Breast Neoplasms
Academic Article	Structural and Functional Analyses of an Allosteric EYA2 Phosphatase Inhibitor That Has On-Target Effects in Human Lung Cancer Cells.
Academic Article	Guidelines and definitions for research on epithelial-mesenchymal transition.
Academic Article	Identification of a Small-Molecule Inhibitor That Disrupts the SIX1/EYA2 Complex, EMT, and Metastasis.
Academic Article	Cellular Plasticity in Breast Cancer Progression and Therapy.
Academic Article	Corrigendum to "The Eya phosphatase: Its unique role in cancer" [Int. J. Biochem. Cell Biol. 96 (2018) 165-170].
Academic Article	VEGF-C mediates tumor growth and metastasis through promoting EMT-epithelial breast cancer cell crosstalk.
Academic Article	Targeting EYA2 tyrosine phosphatase activity in glioblastoma stem cells induces mitotic catastrophe.
Academic Article	SIX1 and EWS/FLI1 co-regulate an anti-metastatic gene network in Ewing Sarcoma.
Academic Article	EYA2 tyrosine phosphatase inhibition reduces MYC and prevents medulloblastoma progression.
Academic Article	Interferon-Induced Bone Marrow Stromal Antigen 2 (BST2) Is A Functional Tumor-Initiating Cell Marker In Triple-Negative Breast Cancer.

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Neoplasms