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Search Results to David A Schwartz

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One or more keywords matched the following items that are connected to Schwartz, David

Item TypeName
Academic Article Endotoxin responsiveness and grain dust-induced inflammation in the lower respiratory tract.
Academic Article Role of endotoxin in grain dust-induced lung inflammation in mice.
Academic Article Pentoxifylline does not alter the response to inhaled grain dust.
Academic Article Cytokine gene expression after inhalation of corn dust.
Academic Article Bacterial DNA or oligonucleotides containing unmethylated CpG motifs can minimize lipopolysaccharide-induced inflammation in the lower respiratory tract through an IL-12-dependent pathway.
Academic Article The effects of inhalation of grain dust extract and endotoxin on upper and lower airways.
Academic Article The role of TLR4 in endotoxin responsiveness in humans.
Academic Article Subchronic endotoxin inhalation causes persistent airway disease.
Academic Article Fibrinolysis in LPS-induced chronic airway disease.
Academic Article The role of Toll-like receptor 4 in environmental airway injury in mice.
Academic Article LPS binding protein is important in the airway response to inhaled endotoxin.
Academic Article The critical role of hematopoietic cells in lipopolysaccharide-induced airway inflammation.
Academic Article Toll-like receptor 4 antagonist (E5564) prevents the chronic airway response to inhaled lipopolysaccharide.
Academic Article The transcriptional response to lipopolysaccharide reveals a role for interferon-gamma in lung neutrophil recruitment.
Academic Article Ambient ozone primes pulmonary innate immunity in mice.
Academic Article Fibroproliferation in LPS-induced airway remodeling and bleomycin-induced fibrosis share common patterns of gene expression.
Academic Article Chronic LPS inhalation causes emphysema-like changes in mouse lung that are associated with apoptosis.
Academic Article Toll-like receptor 4 variants reduce airway response in human subjects at high endotoxin levels in a swine facility.
Academic Article TLR4 is necessary for hyaluronan-mediated airway hyperresponsiveness after ozone inhalation.
Academic Article TLR4 mutations are associated with endotoxin hyporesponsiveness in humans.
Academic Article The genetic and environmental causes of pulmonary fibrosis.
Academic Article Integrating murine gene expression studies to understand obstructive lung disease due to chronic inhaled endotoxin.
Academic Article Grain dust and endotoxin inhalation challenges produce similar inflammatory responses in normal subjects.
Academic Article The effect of of glucocorticoids on grain dust-induced airway disease.
Academic Article Variable airway responsiveness to inhaled lipopolysaccharide.
Academic Article Compartmentalization of the inflammatory response to inhaled grain dust.
Academic Article Bronchial hyperreactivity is associated with enhanced grain dust-induced airflow obstruction.
Academic Article Endotoxin responsiveness and subchronic grain dust-induced airway disease.
Academic Article Does inhalation of endotoxin cause asthma?
Academic Article Airway inflammation and responsiveness in prostaglandin H synthase-deficient mice exposed to bacterial lipopolysaccharide.
Academic Article CD14 is an essential mediator of LPS-induced airway disease.
Academic Article Spontaneous mutations in recombinant inbred mice: mutant toll-like receptor 4 (Tlr4) in BXD29 mice.
Academic Article Safety of incremental inhaled lipopolysaccharide challenge in humans.
Academic Article Alloimmune lung injury induced by local innate immune activation through inhaled lipopolysaccharide.
Academic Article Leukocyte-derived IL-10 reduces subepithelial fibrosis associated with chronically inhaled endotoxin.
Academic Article The pulmonary sequelae associated with accidental inhalation of chlorine gas.
Concept Inhalation
Concept Inhalation Exposure
Concept Administration, Inhalation
Grant GRAIN DUST, ENDOTOXIN, AND AIRFLOW OBSTRUCTION
Grant HOST DETERMINANTS OF ORGANIC DUST INDUCED AIRWAY DISEASE
Grant Environmental Determinants of Airway Disease in Children

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  • Inhalation

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