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Search Results to David W h Riches

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One or more keywords matched the following items that are connected to Riches, David

Item TypeName
Academic Article Human insulin-like growth factor-IA expression in transgenic mice promotes adenomatous hyperplasia but not pulmonary fibrosis.
Academic Article Compartmentalized expression of c-FLIP in lung tissues of patients with idiopathic pulmonary fibrosis.
Academic Article Increased cell surface Fas expression is necessary and sufficient to sensitize lung fibroblasts to Fas ligation-induced apoptosis: implications for fibroblast accumulation in idiopathic pulmonary fibrosis.
Academic Article IL-4-induced macrophage-derived IGF-I protects myofibroblasts from apoptosis following growth factor withdrawal.
Academic Article TNF-alpha sensitizes normal and fibrotic human lung fibroblasts to Fas-induced apoptosis.
Concept Fibroblasts
Concept Fibroblast Growth Factor 10
Concept Receptor, Fibroblast Growth Factor, Type 2
Academic Article The fibroproliferative response in acute respiratory distress syndrome: mechanisms and clinical significance.
Academic Article Protein tyrosine phosphatase a mediates profibrotic signaling in lung fibroblasts through TGF-? responsiveness.
Grant Mechanisms of myofibroblast death in pulmonary fibrosis
Grant Therapeutic Targeting of PTPN13 in Idiopathic Pulmonary Fibrosis
Grant Fibroblast Resistance to Apoptosis in Pulmonary Fibrosis
Academic Article FGF10-FGFR2B Signaling Generates Basal Cells and Drives Alveolar Epithelial Regeneration by Bronchial Epithelial Stem Cells after Lung Injury.
Academic Article Potential of nintedanib in treatment of progressive fibrosing interstitial lung diseases.
Academic Article Protein tyrosine phosphatase-a amplifies transforming growth factor-?-dependent profibrotic signaling in lung fibroblasts.
Academic Article Loss of Fas signaling in fibroblasts impairs homeostatic fibrosis resolution and promotes persistent pulmonary fibrosis.
Academic Article Alveolar epithelial cells and microenvironmental stiffness synergistically drive fibroblast activation in three-dimensional hydrogel lung models.
Academic Article Inhibition of antiapoptotic BCL-2 proteins with ABT-263 induces fibroblast apoptosis, reversing persistent pulmonary fibrosis.

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  • Fibroblasts

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