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Search Results to Christian D Young

This is a "connection" page, showing the details of why an item matched the keywords from your search.

                     
                     

One or more keywords matched the following items that are connected to Young, Christian

Item TypeName
Academic Article AKT regulates BRCA1 stability in response to hormone signaling.
Concept Phosphatidylinositols
Concept Phosphatidylinositol 3-Kinases
Academic Article Conditional loss of ErbB3 delays mammary gland hyperplasia induced by mutant PIK3CA without affecting mammary tumor latency, gene expression, or signaling.
Academic Article Role of EphB3 Receptor in Mediating Head and Neck Tumor Growth, Cell Migration, and Response to PI3K Inhibitor.
Concept Class I Phosphatidylinositol 3-Kinases
Concept Phosphatidylinositol 3-Kinase
Academic Article Cancer Cell CD44 Mediates Macrophage/Monocyte-Driven Regulation of Head and Neck Cancer Stem Cells.
Academic Article Deletion of p53 and Hyper-Activation of PIK3CA in Keratin-15+ Stem Cells Lead to the Development of Spontaneous Squamous Cell Carcinoma.
Academic Article Activating PIK3CA Mutations Induce an Epidermal Growth Factor Receptor (EGFR)/Extracellular Signal-regulated Kinase (ERK) Paracrine Signaling Axis in Basal-like Breast Cancer.
Academic Article The receptor tyrosine kinase ErbB3 maintains the balance between luminal and basal breast epithelium.
Academic Article Mutant PIK3CA accelerates HER2-driven transgenic mammary tumors and induces resistance to combinations of anti-HER2 therapies.
Academic Article HER3 is required for HER2-induced preneoplastic changes to the breast epithelium and tumor formation.
Academic Article ErbB3 ablation impairs PI3K/Akt-dependent mammary tumorigenesis.
Academic Article Epidermal growth factor receptor signaling in precancerous keratinocytes promotes neighboring head and neck cancer squamous cell carcinoma cancer stem cell-like properties and phosphoinositide 3-kinase inhibitor insensitivity.
Academic Article Polymorphonuclear myeloid-derived suppressor cells and phosphatidylinositol-3 kinase gamma are critical to tobacco-mimicking oral carcinogenesis in mice.

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  • Phosphatidylinositols

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