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MatchTypeWhy
Rewers, MarianPerson Why?
Norris, JillPerson Why?
Holers, VernonPerson Why?
Islet Autoimmunity Reversion as a Model of T1D ResiliencyGrant Why?
Changes in the Coexpression of Innate Immunity Genes During Persistent Islet Autoimmunity Are Associated With Progression of Islet Autoimmunity: Diabetes Autoimmunity Study in the Young (DAISY).Academic Article Why?
Autoimmunity risk alleles compromising B cell anergyGrant Why?
Early childhood infections and the risk of islet autoimmunity: the Diabetes Autoimmunity Study in the Young (DAISY).Academic Article Why?
Late-onset islet autoimmunity in childhood: the Diabetes Autoimmunity Study in the Young (DAISY).Academic Article Why?
Longitudinal changes in DNA methylation during the onset of islet autoimmunity differentiate between reversion versus progression of islet autoimmunity.Academic Article Why?
Normal but increasing hemoglobin A1c levels predict progression from islet autoimmunity to overt type 1 diabetes: Diabetes Autoimmunity Study in the Young (DAISY).Academic Article Why?
Phospholipid Levels at Seroconversion Are Associated With Resolution of Persistent Islet Autoimmunity: The Diabetes Autoimmunity Study in the Young.Academic Article Why?
Somatic mutagenesis in autoimmunity.Academic Article Why?
Deane, KevinPerson Why?
A PD-1-specific toxin suppresses autoimmunity.Academic Article Why?
Absence of functional fetal regulatory T cells in humans causes in utero organ-specific autoimmunity.Academic Article Why?
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