Amitrole
"Amitrole" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A non-selective post-emergence, translocated herbicide. According to the Seventh Annual Report on Carcinogens (PB95-109781, 1994) this substance may reasonably be anticipated to be a carcinogen. (From Merck Index, 12th ed) It is an irreversible inhibitor of CATALASE, and thus impairs activity of peroxisomes.
Descriptor ID |
D000640
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MeSH Number(s) |
D03.383.129.799.100
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Concept/Terms |
Amitrole- Amitrole
- 3-Amino-1,2,4-triazole
- Aminotriazole
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Below are MeSH descriptors whose meaning is more general than "Amitrole".
Below are MeSH descriptors whose meaning is more specific than "Amitrole".
This graph shows the total number of publications written about "Amitrole" by people in this website by year, and whether "Amitrole" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
Year | Major Topic | Minor Topic | Total |
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2004 | 0 | 1 | 1 |
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Below are the most recent publications written about "Amitrole" by people in Profiles.
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Carter AB, Tephly LA, Venkataraman S, Oberley LW, Zhang Y, Buettner GR, Spitz DR, Hunninghake GW. High levels of catalase and glutathione peroxidase activity dampen H2O2 signaling in human alveolar macrophages. Am J Respir Cell Mol Biol. 2004 Jul; 31(1):43-53.
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Banerjee A, Grosso MA, Brown JM, Rogers KB, Whitman GJ. Oxygen metabolite effects on creatine kinase and cardiac energetics after reperfusion. Am J Physiol. 1991 Aug; 261(2 Pt 2):H590-7.
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Berger EM, Beehler CJ, Harada RN, Repine JE. Human phagocytic cells as oxygen metabolite scavengers. Inflammation. 1990 Oct; 14(5):613-9.
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Brown JM, White CW, Terada LS, Grosso MA, Shanley PF, Mulvin DW, Banerjee A, Whitman GJ, Harken AH, Repine JE. Interleukin 1 pretreatment decreases ischemia/reperfusion injury. Proc Natl Acad Sci U S A. 1990 Jul; 87(13):5026-30.
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McCutchan HJ, Schwappach JR, Enquist EG, Walden DL, Terada LS, Reiss OK, Leff JA, Repine JE. Xanthine oxidase-derived H2O2 contributes to reperfusion injury of ischemic skeletal muscle. Am J Physiol. 1990 May; 258(5 Pt 2):H1415-9.
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Brown JM, Grosso MA, Terada LS, Whitman GJ, Banerjee A, White CW, Harken AH, Repine JE. Endotoxin pretreatment increases endogenous myocardial catalase activity and decreases ischemia-reperfusion injury of isolated rat hearts. Proc Natl Acad Sci U S A. 1989 Apr; 86(7):2516-20.
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Brown JM, Grosso MA, Whitman GJ, Banerjee A, Terada LS, Repine JE, Harken AH. The coincidence of myocardial reperfusion injury and hydrogen peroxide production in the isolated rat heart. Surgery. 1989 Apr; 105(4):496-501.
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Brown JM, Grosso MA, Banerjee A, Whitman GJ, Repine JE, Harken AH. Erythrocyte catalase decreases cardiac reperfusion injury. Curr Surg. 1989 Mar-Apr; 46(2):122-4.
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Brown JM, Terada LS, Grosso MA, Whitman GJ, Velasco SE, Patt A, Harken AH, Repine JE. Hydrogen peroxide mediates reperfusion injury in the isolated rat heart. Mol Cell Biochem. 1988 Dec; 84(2):173-5.
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Brown JM, Terada LS, Grosso MA, Whitmann GJ, Velasco SE, Patt A, Harken AH, Repine JE. Xanthine oxidase produces hydrogen peroxide which contributes to reperfusion injury of ischemic, isolated, perfused rat hearts. J Clin Invest. 1988 Apr; 81(4):1297-301.
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