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Search Results to Douglas R Seals

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One or more keywords matched the following items that are connected to Seals, Douglas

Item TypeName
Academic Article Habitually exercising older men do not demonstrate age-associated vascular endothelial oxidative stress.
Academic Article MicroRNA changes in human arterial endothelial cells with senescence: relation to apoptosis, eNOS and inflammation.
Academic Article Aging is associated with greater nuclear NF kappa B, reduced I kappa B alpha, and increased expression of proinflammatory cytokines in vascular endothelial cells of healthy humans.
Academic Article Nitrite supplementation reverses vascular endothelial dysfunction and large elastic artery stiffness with aging.
Academic Article Short-term calorie restriction reverses vascular endothelial dysfunction in old mice by increasing nitric oxide and reducing oxidative stress.
Academic Article Replicative aging induces endothelial to mesenchymal transition in human aortic endothelial cells: potential role of inflammation.
Academic Article Superoxide-lowering therapy with TEMPOL reverses arterial dysfunction with aging in mice.
Concept Cellular Senescence
Academic Article Life-long caloric restriction reduces oxidative stress and preserves nitric oxide bioavailability and function in arteries of old mice.
Academic Article Superoxide signaling in perivascular adipose tissue promotes age-related artery stiffness.
Academic Article Nicotinamide mononucleotide supplementation reverses vascular dysfunction and oxidative stress with aging in mice.
Academic Article Dietary rapamycin supplementation reverses age-related vascular dysfunction and oxidative stress, while modulating nutrient-sensing, cell cycle, and senescence pathways.
Academic Article Endothelial cell senescence with aging in healthy humans: prevention by habitual exercise and relation to vascular endothelial function.
Grant Role of cellular senescence in cardiovascular aging
Academic Article Short-term interleukin-37 treatment improves vascular endothelial function, endurance exercise capacity, and whole-body glucose metabolism in old mice.

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  • Cellular Senescence

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