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Search Results to David A Schwartz

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Schwartz, David

One or more keywords matched the following items that are connected to Schwartz, David

Item Type	Name
Academic Article	The effects of inhalation of grain dust extract and endotoxin on upper and lower airways.
Academic Article	Subchronic endotoxin inhalation causes persistent airway disease.
Academic Article	Ozone-induced acute pulmonary injury in inbred mouse strains.
Academic Article	Altered surfactant protein A gene expression and protein metabolism associated with repeat exposure to inhaled endotoxin.
Academic Article	Fibroproliferation in LPS-induced airway remodeling and bleomycin-induced fibrosis share common patterns of gene expression.
Academic Article	TLR4 mutations are associated with endotoxin hyporesponsiveness in humans.
Academic Article	The clinical and environmental determinants of airway transcriptional profiles in allergic asthma.
Academic Article	Detrimental role of the airway mucin Muc5ac during ventilator-induced lung injury.
Academic Article	The idiopathic pulmonary fibrosis honeycomb cyst contains a mucocilary pseudostratified epithelium.
Academic Article	Compartmentalization of the inflammatory response to inhaled grain dust.
Academic Article	The genetics of innate immunity.
Academic Article	Leukocyte-derived IL-10 reduces subepithelial fibrosis associated with chronically inhaled endotoxin.
Concept	Intestinal Mucosa
Concept	Mucous Membrane
Concept	Nasal Mucosa
Concept	Respiratory Mucosa
Academic Article	Muc5b is required for airway defence.
Academic Article	Future directions in idiopathic pulmonary fibrosis research. An NHLBI workshop report.
Academic Article	Idiopathic Pulmonary Fibrosis: A Genetic Disease That Involves Mucociliary Dysfunction of the Peripheral Airways.
Academic Article	The nasal methylome and childhood atopic asthma.
Academic Article	The Nasal Methylome: A Key to Understanding Allergic Asthma.
Academic Article	Idiopathic Pulmonary Fibrosis Is a Genetic Disease Involving Mucus and the Peripheral Airways.
Academic Article	Muc5b overexpression causes mucociliary dysfunction and enhances lung fibrosis in mice.
Academic Article	Revealing the Secrets of Idiopathic Pulmonary Fibrosis.
Academic Article	ILC2s mediate systemic innate protection by priming mucus production at distal mucosal sites.
Academic Article	Genes, other than Muc5b, play a role in bleomycin-induced lung fibrosis.

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Mucous Membrane