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Search Results to Mark L Dell'Acqua

This is a "connection" page, showing the details of why an item matched the keywords from your search.


One or more keywords matched the following items that are connected to Dell'Acqua, Mark

Item TypeName
Academic Article Regulation of postsynaptic structure and function by an A-kinase anchoring protein-membrane-associated guanylate kinase scaffolding complex.
Academic Article Palmitoylation of A-kinase anchoring protein 79/150 regulates dendritic endosomal targeting and synaptic plasticity mechanisms.
Academic Article AKAP150-anchored calcineurin regulates synaptic plasticity by limiting synaptic incorporation of Ca2+-permeable AMPA receptors.
Academic Article Phospholipase C is required for changes in postsynaptic structure and function associated with NMDA receptor-dependent long-term depression.
Concept Dendritic Spines
Academic Article AKAP-anchored PKA maintains neuronal L-type calcium channel activity and NFAT transcriptional signaling.
Academic Article The palmitoyl acyltransferase DHHC2 regulates recycling endosome exocytosis and synaptic potentiation through palmitoylation of AKAP79/150.
Academic Article Inhibition of the Motor Protein Eg5/Kinesin-5 in Amyloid ß-Mediated Impairment of Hippocampal Long-Term Potentiation and Dendritic Spine Loss.
Academic Article STIM1 Ca2+ Sensor Control of L-type Ca2+-Channel-Dependent Dendritic Spine Structural Plasticity and Nuclear Signaling.
Grant Regulation of Calcium-permeable AMPA Receptors by AKAP79 Postsynaptic Signaling
Grant Amyloid Beta Postsynaptic Signaling through AKAP-anchored Calcineurin
Academic Article AKAP150 Palmitoylation Regulates Synaptic Incorporation of Ca2+-Permeable AMPA Receptors to Control LTP.
Academic Article AKAP79/150 recruits the transcription factor NFAT to regulate signaling to the nucleus by neuronal L-type Ca2+ channels.
Academic Article Synaptic crosstalk conferred by a zone of differentially regulated Ca2+ signaling in the dendritic shaft adjoining a potentiated spine.
Grant L-type Ca2+ Channel Spike Regulation of Spine Structural Plasticity and Excitation-Transcription Coupling

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  • Dendritic Spines

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