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Search Results to Karl Ulrich Bayer

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Bayer, Karl

One or more keywords matched the following items that are connected to Bayer, Karl

Item Type	Name
Academic Article	Regulation of signal transduction by protein targeting: the case for CaMKII.
Academic Article	Transition from reversible to persistent binding of CaMKII to postsynaptic sites and NR2B.
Academic Article	Role of the CaMKII/NMDA receptor complex in the maintenance of synaptic strength.
Academic Article	Selective regulation of neurite extension and synapse formation by the beta but not the alpha isoform of CaMKII.
Academic Article	Selective translocation of Ca2+/calmodulin protein kinase IIalpha (CaMKIIalpha) to inhibitory synapses.
Academic Article	CaMKII regulation in information processing and storage.
Concept	Synapses
Concept	Electrical Synapses
Academic Article	Enzymatic activity of CaMKII is not required for its interaction with the glutamate receptor subunit GluN2B.
Academic Article	Necessary, but not sufficient: insights into the mechanisms of mGluR mediated long-term depression from a rat model of early life seizures.
Academic Article	Autonomous CaMKII requires further stimulation by Ca2+/calmodulin for enhancing synaptic strength.
Academic Article	NMDA receptor activation strengthens weak electrical coupling in mammalian brain.
Academic Article	The CaMKII/GluN2B Protein Interaction Maintains Synaptic Strength.
Grant	CaMKII in global cerebral ischemia
Academic Article	CaMKII regulates the depalmitoylation and synaptic removal of the scaffold protein AKAP79/150 to mediate structural long-term depression.
Grant	Structural and Catalytic Functions of CaMKII in Neurons
Grant	Cell-permeable inibitors specific for CaMKII
Academic Article	Simultaneous Live Imaging of Multiple Endogenous Proteins Reveals a Mechanism for Alzheimer's-Related Plasticity Impairment.
Academic Article	The CaMKII K42M and K42R mutations are equivalent in suppressing kinase activity and targeting.
Academic Article	CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity.
Academic Article	Stimulating ?-adrenergic receptors promotes synaptic potentiation by switching CaMKII movement from LTD to LTP mode.
Academic Article	Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in mice.

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Synapses