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Search Results to Karl Ulrich Bayer

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Bayer, Karl

One or more keywords matched the following items that are connected to Bayer, Karl

Item Type	Name
Academic Article	Interaction with the NMDA receptor locks CaMKII in an active conformation.
Academic Article	Regulation of signal transduction by protein targeting: the case for CaMKII.
Academic Article	Transition from reversible to persistent binding of CaMKII to postsynaptic sites and NR2B.
Academic Article	CaMKIIbeta association with the actin cytoskeleton is regulated by alternative splicing.
Academic Article	CaMKII "autonomy" is required for initiating but not for maintaining neuronal long-term information storage.
Academic Article	Improving a natural CaMKII inhibitor by random and rational design.
Academic Article	Alternative splicing modulates the frequency-dependent response of CaMKII to Ca(2+) oscillations.
Academic Article	Calcium/calmodulin-dependent protein kinase II binds to Raf-1 and modulates integrin-stimulated ERK activation.
Academic Article	Dual mechanism of a natural CaMKII inhibitor.
Academic Article	CaMKII autonomy is substrate-dependent and further stimulated by Ca2+/calmodulin.
Academic Article	Differential regulation by ATP versus ADP further links CaMKII aggregation to ischemic conditions.
Academic Article	Calmodulin-dependent kinase II mediates vascular smooth muscle cell proliferation and is potentiated by extracellular signal regulated kinase.
Academic Article	Nucleotides and phosphorylation bi-directionally modulate Ca2+/calmodulin-dependent protein kinase II (CaMKII) binding to the N-methyl-D-aspartate (NMDA) receptor subunit GluN2B.
Academic Article	A significant but rather mild contribution of T286 autophosphorylation to Ca2+/CaM-stimulated CaMKII activity.
Academic Article	CaMKII regulation in information processing and storage.
Concept	Phosphorylation
Academic Article	Camkii-mediated phosphorylation regulates distributions of Syngap-a1 and -a2 at the postsynaptic density.
Academic Article	Enzymatic activity of CaMKII is not required for its interaction with the glutamate receptor subunit GluN2B.
Academic Article	Autonomous CaMKII mediates both LTP and LTD using a mechanism for differential substrate site selection.
Academic Article	CaMKII mediates recruitment and activation of the deubiquitinase CYLD at the postsynaptic density.
Academic Article	CaMKII binding to GluN2B is differentially affected by macromolecular crowding reagents.
Academic Article	Autonomous CaMKII requires further stimulation by Ca2+/calmodulin for enhancing synaptic strength.
Academic Article	Nitric oxide induces Ca2+-independent activity of the Ca2+/calmodulin-dependent protein kinase II (CaMKII).
Academic Article	CaMKII-mediated displacement of AIDA-1 out of the postsynaptic density core.
Academic Article	Autonomous CaMKII Activity as a Drug Target for Histological and Functional Neuroprotection after Resuscitation from Cardiac Arrest.
Grant	CaMKII in global cerebral ischemia
Academic Article	The CaMKII holoenzyme structure in activation-competent conformations.
Academic Article	DAPK1 Mediates LTD by Making CaMKII/GluN2B Binding LTP Specific.
Academic Article	CaMKII Metaplasticity Drives A? Oligomer-Mediated Synaptotoxicity.
Grant	Cell-permeable inibitors specific for CaMKII
Academic Article	CaM Kinase: Still Inspiring at 40.
Academic Article	The CaMKII K42M and K42R mutations are equivalent in suppressing kinase activity and targeting.
Academic Article	CaMKIIa knockout protects from ischemic neuronal cell death after resuscitation from cardiac arrest.
Academic Article	GluN2B S1303 phosphorylation by CaMKII or DAPK1: no indication for involvement in ischemia or LTP.
Academic Article	Conserved and divergent features of neuronal CaMKII holoenzyme structure, function, and high-order assembly.
Academic Article	CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity.
Academic Article	Short-term CaMKII inhibition with tatCN19o does not erase pre-formed memory in mice and is neuroprotective in pigs.
Academic Article	Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in mice.
Academic Article	LTP induction by structural rather than enzymatic functions of CaMKII.

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Phosphorylation