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Search Results to Raul Martin Torres

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Torres, Raul

One or more keywords matched the following items that are connected to Torres, Raul

Item Type	Name
Academic Article	Receptor editing is the main mechanism of B cell tolerance toward membrane antigens.
Academic Article	Receptor editing can lead to allelic inclusion and development of B cells that retain antibodies reacting with high avidity autoantigens.
Academic Article	Receptor editing for better or for worse.
Academic Article	Arhgef1 is required by T cells for the development of airway hyperreactivity and inflammation.
Academic Article	Ig allotypic inclusion does not prevent B cell development or response.
Academic Article	TLR agonists promote marginal zone B cell activation and facilitate T-dependent IgM responses.
Academic Article	Lsc activity is controlled by oligomerization and regulates integrin adhesion.
Academic Article	Arhgef1 regulates alpha5beta1 integrin-mediated matrix metalloproteinase expression and is required for homeostatic lung immunity.
Academic Article	BAFF receptor signaling aids the differentiation of immature B cells into transitional B cells following tonic BCR signaling.
Academic Article	Central B-cell tolerance: where selection begins.
Academic Article	Antigen and cytokine receptor signals guide the development of the na?ve mature B cell repertoire.
Academic Article	Studies of lymphocyte reconstitution in a humanized mouse model reveal a requirement of T cells for human B cell maturation.
Academic Article	IL-23-dependent IL-17 production is essential in neutrophil recruitment and activity in mouse lung defense against respiratory Mycoplasma pneumoniae infection.
Academic Article	Lsc regulates marginal-zone B cell migration and adhesion and is required for the IgM T-dependent antibody response.
Academic Article	Ras activation of Erk restores impaired tonic BCR signaling and rescues immature B cell differentiation.
Academic Article	S1P3 confers differential S1P-induced migration by autoreactive and non-autoreactive immature B cells and is required for normal B-cell development.
Academic Article	Type I IFN enhances follicular B cell contribution to the T cell-independent antibody response.
Academic Article	Division of labor during primary humoral immunity.
Academic Article	Mouse marginal zone B cells harbor specificities similar to human broadly neutralizing HIV antibodies.
Academic Article	Murine B cell development and antibody responses to model antigens are not impaired in the absence of the TNF receptor GITR.
Academic Article	Murine gammaherpesvirus 68 infection protects lupus-prone mice from the development of autoimmunity.
Academic Article	Dual-reactive B cells are autoreactive and highly enriched in the plasmablast and memory B cell subsets of autoimmune mice.
Concept	Precursor Cells, B-Lymphoid
Concept	T-Lymphocytes, Cytotoxic
Concept	CD8-Positive T-Lymphocytes
Concept	T-Lymphocytes, Helper-Inducer
Concept	Dendritic Cells
Concept	CD4-Positive T-Lymphocytes
Concept	Antigen-Presenting Cells
Concept	Th2 Cells
Concept	B-Lymphocytes
Concept	Bone Marrow Cells
Concept	Plasma Cells
Concept	Cells, Cultured
Concept	T-Lymphocytes
Academic Article	Lysophosphatidic acid inhibits CD8 T cell activation and control of tumor progression.
Academic Article	Lysophosphatidic acid receptor 5 inhibits B cell antigen receptor signaling and antibody response.
Academic Article	Activation of Ras overcomes B-cell tolerance to promote differentiation of autoreactive B cells and production of autoantibodies.
Academic Article	Receptor editing and genetic variability in human autoreactive B cells.
Academic Article	Mechanism of Mitochondrial Transcription Factor A Attenuation of CpG-Induced Antibody Production.
Academic Article	A Special Connection between ?d T Cells and Natural Antibodies?
Grant	Antigen and lysophospholipid receptor regulation of lymphocyte development and fu
Grant	Marginal Zone B Cell Response to HIV
Academic Article	Immunological tolerance as a barrier to protective HIV humoral immunity.
Academic Article	Activation of the MEK-ERK Pathway Is Necessary but Not Sufficient for Breaking Central B Cell Tolerance.
Academic Article	Immune Surveillance by Natural IgM Is Required for Early Neoantigen Recognition and Initiation of Adaptive Immunity.
Grant	Humoral Immunity by Anergic B cells
Academic Article	Silencing of TLM B cells by chronic HIV infection.
Academic Article	Innate and adaptive signals enhance differentiation and expansion of dual-antibody autoreactive B cells in lupus.
Academic Article	Active PI3K abrogates central tolerance in high-avidity autoreactive B cells.
Academic Article	LPA5 Is an Inhibitory Receptor That Suppresses CD8 T-Cell Cytotoxic Function via Disruption of Early TCR Signaling.
Academic Article	Ushering along B cells to neutralize HIV.
Grant	Lysophosphatidic Acid Regulation of CD8 T cell activation and function
Academic Article	Lysophosphatidic Acid Is an Inflammatory Lipid Exploited by Cancers for Immune Evasion via Mechanisms Similar and Distinct From CTLA-4 and PD-1.
Academic Article	B-cell intrinsic and extrinsic signals that regulate central tolerance of mouse and human B cells.
Academic Article	Elevated Detection of Dual Antibody B Cells Identifies Lupus Patients With B Cell-Reactive VH4-34 Autoantibodies.
Academic Article	SARS-CoV-2 infection relaxes peripheral B cell tolerance.
Academic Article	LPA suppresses T cell function by altering the cytoskeleton and disrupting immune synapse formation.
Academic Article	Autoantibodies elicited with SARS-CoV-2 infection are linked to alterations in double negative B cells.
Academic Article	CD19 Is Internalized Together with IgM in Proportion to B Cell Receptor Stimulation and Is Modulated by Phosphatidylinositol 3-Kinase in Bone Marrow Immature B Cells.
Academic Article	Regulation of CD8 T-cell signaling, metabolism, and cytotoxic activity by extracellular lysophosphatidic acid.
Academic Article	Lysophosphatidic acid modulates CD8 T cell immunosurveillance and metabolism to impair anti-tumor immunity.
Academic Article	Activation and pro-inflammatory cytokine production by unswitched memory B cells during SARS-CoV-2 infection.

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