Colorado PROFILES, The Colorado Clinical and Translational Sciences Institute (CCTSI)
Keywords
Last Name
Institution

Contact Us
If you have any questions or feedback please contact us.

Search Results (276)

Click the Why column to see why an item matched the search.

MatchTypeWhy
Astrocyte inflammatory signaling mediates a-synuclein aggregation and dopaminergic neuronal loss following viral encephalitis.Academic Article Why?
Blood-brain barrier resealing in neuromyelitis optica occurs independently of astrocyte regeneration.Academic Article Why?
The role of astrocytes in inhibitory synaptogenesisGrant Why?
Astrocytes Underlie Neuroinflammatory Memory Impairment.Academic Article Why?
Calmodulin kinase II in pure cultured astrocytes.Academic Article Why?
Common molecular mechanisms of SLC6A1 variant-mediated neurodevelopmental disorders in astrocytes and neurons.Academic Article Why?
Humoral Immunity, Astrocyte Injury, and Demyelination in Neuromyelitis OpticaGrant Why?
Neuron-specific mitochondrial oxidative stress results in epilepsy, glucose dysregulation and a striking astrocyte response.Academic Article Why?
Striatal neuronal death mediated by astrocytes from the Gcdh-/- mouse model of glutaric acidemia type I.Academic Article Why?
Astrocyte Glutamate Uptake and Water Homeostasis Are Dysregulated in the Hippocampus of Multiple Sclerosis Patients With Seizures.Academic Article Why?
Toxoplasma gondii invasion and replication in astrocyte primary cultures and astrocytoma cell lines: systematic review of the literature.Academic Article Why?
Watkins, LindaPerson Why?
AstrocytesConcept Why?
TDP-43 knockdown causes innate immune activation via protein kinase R in astrocytes.Academic Article Why?
Varicella-Zoster Virus Infection of Primary Human Spinal Astrocytes Produces Intracellular Amylin, Amyloid-ß, and an Amyloidogenic Extracellular Environment.Academic Article Why?
Per Page    Page  of 19last Nextnext
Prev
Search Criteria
  • astrocytes
Filter by Type

Copyright © 2022 The Regents of the University of Colorado, a body corporate. All rights reserved. (Harvard PROFILES RNS software version: 2.11.1)