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MatchTypeWhy
Rewers, MarianPerson Why?
Norris, JillPerson Why?
Holers, VernonPerson Why?
Cambier, JohnPerson Why?
Islet Autoimmunity Reversion as a Model of T1D ResiliencyGrant Why?
Changes in the Coexpression of Innate Immunity Genes During Persistent Islet Autoimmunity Are Associated With Progression of Islet Autoimmunity: Diabetes Autoimmunity Study in the Young (DAISY).Academic Article Why?
Deane, KevinPerson Why?
Autoimmunity risk alleles compromising B cell anergyGrant Why?
Early childhood infections and the risk of islet autoimmunity: the Diabetes Autoimmunity Study in the Young (DAISY).Academic Article Why?
Late-onset islet autoimmunity in childhood: the Diabetes Autoimmunity Study in the Young (DAISY).Academic Article Why?
Normal but increasing hemoglobin A1c levels predict progression from islet autoimmunity to overt type 1 diabetes: Diabetes Autoimmunity Study in the Young (DAISY).Academic Article Why?
Phospholipid Levels at Seroconversion Are Associated With Resolution of Persistent Islet Autoimmunity: The Diabetes Autoimmunity Study in the Young.Academic Article Why?
Somatic mutagenesis in autoimmunity.Academic Article Why?
A PD-1-specific toxin suppresses autoimmunity.Academic Article Why?
Autoimmunity-associated T cell receptors recognize HLA-B*27-bound peptides.Academic Article Why?
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