Rescue of deficient amygdala tonic ?-aminobutyric acidergic currents in the Fmr-/y mouse model of fragile X syndrome by a novel ?-aminobutyric acid type A receptor-positive allosteric modulator.

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Rescue of deficient amygdala tonic ?-aminobutyric acidergic currents in the Fmr-/y mouse model of fragile X syndrome by a novel ?-aminobutyric acid type A receptor-positive allosteric modulator.

Martin BS, Martinez-Botella G, Loya CM, Salituro FG, Robichaud AJ, Huntsman MM, Ackley MA, Doherty JJ, Corbin JG. Rescue of deficient amygdala tonic ?-aminobutyric acidergic currents in the Fmr-/y mouse model of fragile X syndrome by a novel ?-aminobutyric acid type A receptor-positive allosteric modulator. J Neurosci Res. 2016 Jun; 94(6):568-78.

View in: PubMed

subject areas

Amygdala
Animals
Animals, Newborn
CHO Cells
Cricetulus
Disease Models, Animal
Dose-Response Relationship, Drug
Fragile X Mental Retardation Protein
Fragile X Syndrome
GABA Agents
GABA Modulators
gamma-Aminobutyric Acid
Heterocyclic Compounds, 2-Ring
In Vitro Techniques
Membrane Potentials
Mice
Mice, Knockout
Patch-Clamp Techniques
Pregnanolone
Receptors, GABA-A
Transfection

authors with profiles

Molly-Maureen Huntsman

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