 Mice, Inbred C57BL
"Mice, Inbred C57BL" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
One of the first INBRED MOUSE STRAINS to be sequenced. This strain is commonly used as genetic background for transgenic mouse models. Refractory to many tumors, this strain is also preferred model for studying role of genetic variations in development of diseases.
| Descriptor ID |
D008810
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| MeSH Number(s) |
B01.050.050.199.520.520.420 B01.050.150.900.649.313.992.635.505.500.400.420
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| Concept/Terms |
Mice, Inbred C57BL- Mice, Inbred C57BL
- C57BL Mice, Inbred
- Inbred C57BL Mice
- Mouse, Inbred C57BL
- C57BL Mouse, Inbred
- Inbred C57BL Mouse
- Mice, C57BL
- C57BL Mice
- Mouse, C57BL
- C57BL Mouse
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Below are MeSH descriptors whose meaning is more general than "Mice, Inbred C57BL".
Below are MeSH descriptors whose meaning is more specific than "Mice, Inbred C57BL".
This graph shows the total number of publications written about "Mice, Inbred C57BL" by people in this website by year, and whether "Mice, Inbred C57BL" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1991 | 0 | 4 | 4 | | 1992 | 0 | 17 | 17 | | 1993 | 0 | 16 | 16 | | 1994 | 0 | 16 | 16 | | 1995 | 0 | 14 | 14 | | 1996 | 1 | 26 | 27 | | 1997 | 0 | 21 | 21 | | 1998 | 0 | 27 | 27 | | 1999 | 0 | 37 | 37 | | 2000 | 1 | 47 | 48 | | 2001 | 0 | 65 | 65 | | 2002 | 0 | 81 | 81 | | 2003 | 0 | 84 | 84 | | 2004 | 0 | 74 | 74 | | 2005 | 0 | 99 | 99 | | 2006 | 0 | 119 | 119 | | 2007 | 0 | 124 | 124 | | 2008 | 0 | 156 | 156 | | 2009 | 0 | 137 | 137 | | 2010 | 1 | 157 | 158 | | 2011 | 1 | 194 | 195 | | 2012 | 0 | 178 | 178 | | 2013 | 0 | 196 | 196 | | 2014 | 0 | 204 | 204 | | 2015 | 0 | 174 | 174 | | 2016 | 2 | 169 | 171 | | 2017 | 0 | 171 | 171 | | 2018 | 0 | 166 | 166 | | 2019 | 0 | 175 | 175 | | 2020 | 0 | 95 | 95 | | 2021 | 0 | 7 | 7 |
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Below are the most recent publications written about "Mice, Inbred C57BL" by people in Profiles.
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Baxter BD, Larson ED, Merle L, Feinstein P, Polese AG, Bubak AN, Niemeyer CS, Hassell J, Shepherd D, Ramakrishnan VR, Nagel MA, Restrepo D. Transcriptional profiling reveals potential involvement of microvillous TRPM5-expressing cells in viral infection of the olfactory epithelium. BMC Genomics. 2021 Mar 30; 22(1):224.
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Bithi N, Link C, Henderson YO, Kim S, Yang J, Li L, Wang R, Willard B, Hine C. Dietary restriction transforms the mammalian protein persulfidome in a tissue-specific and cystathionine ?-lyase-dependent manner. Nat Commun. 2021 03 19; 12(1):1745.
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Cai Y, Nielsen BE, Boxer EE, Aoto J, Ford CP. Loss of nigral excitation of cholinergic interneurons contributes to parkinsonian motor impairments. Neuron. 2021 04 07; 109(7):1137-1149.e5.
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Li Y, Gao J, Kamran M, Harmacek L, Danhorn T, Leach SM, O'Connor BP, Hagman JR, Huang H. GATA2 regulates mast cell identity and responsiveness to antigenic stimulation by promoting chromatin remodeling at super-enhancers. Nat Commun. 2021 01 21; 12(1):494.
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Andres-Hernando A, Jensen TJ, Kuwabara M, Orlicky DJ, Cicerchi C, Li N, Roncal-Jimenez CA, Garcia GE, Ishimoto T, Maclean PS, Bjornstad P, Sanchez-Lozada LG, Kanbay M, Nakagawa T, Johnson RJ, Lanaspa MA. Vasopressin mediates fructose-induced metabolic syndrome by activating the V1b receptor. JCI Insight. 2021 01 11; 6(1).
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Mundo W, Wolfson G, Moore LG, Houck JA, Park D, Julian CG. Hypoxia-induced inhibition of mTORC1 activity in the developing lung: a possible mechanism for the developmental programming of pulmonary hypertension. Am J Physiol Heart Circ Physiol. 2021 03 01; 320(3):H980-H990.
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Blaine J. Generating a Podocyte-Specific Neonatal F Receptor (FcRn) Knockout Mouse. Methods Mol Biol. 2021; 2224:123-132.
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Wong H, Levenga J, LaPlante L, Keller B, Cooper-Sansone A, Borski C, Milstead R, Ehringer M, Hoeffer C. Isoform-specific roles for AKT in affective behavior, spatial memory, and extinction related to psychiatric disorders. Elife. 2020 12 16; 9.
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Feseha S, Timic Stamenic T, Wallace D, Tamag C, Yang L, Pan JQ, Todorovic SM. Global genetic deletion of CaV3.3 channels facilitates anaesthetic induction and enhances isoflurane-sparing effects of T-type calcium channel blockers. Sci Rep. 2020 12 09; 10(1):21510.
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Redente EF, Chakraborty S, Sajuthi S, Black BP, Edelman BL, Seibold MA, Riches DW. Loss of Fas signaling in fibroblasts impairs homeostatic fibrosis resolution and promotes persistent pulmonary fibrosis. JCI Insight. 2020 12 08; 6(1).
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